After infection with T. gondii in C2C12 SkMCs, IFN-γ stimulation significantly inhibits parasite growth via inducing IRGs and NO, suggesting that IFN-γ also plays a pivotal role against T. gondii in skeletal cells (Takacs et al., 2012; Wohlfert et al., 2017). The gene discussed is IFNG; the disease is infection.