Similarly, the INHBA+ macrophage subset co-expressed pro-tumorigenic molecules such as IL6, TGFβ, TIMP1, CCL20, CXCL1, and IL10, highlighting the highly plastic and complex nature of tumor-infiltrating myeloid cells in vivo, consistent with recent similar studies in other solid tumors (41, 42, 61–70). The gene discussed is INHBA; the disease is neoplasm.