Th2 response could be promoted by repeated exposure to an antigen (annexin A11 and galactin‐3 have both been recently implicated27), which is at the origin of the theory of an allergic mechanism, supported by the existence of an atopic background in patients, as well as the frequent presence of eosinophilia and elevated IgE.28 Here, IGHE is linked to Increased total eosinophil count.