C-myb is an evolutionarily conserved miR-150 target, and studies have shown that the miR-150/c-myb interaction plays important roles in the differentiation of T cells and B cells, pressure overload-induced cardiac fibrosis, and the regulation of epithelial-mesenchymal transition (EMT) in ovarian cancer cells [24]. The gene discussed is MYB; the disease is ovarian carcinoma.