Since EEI was enriched in interruptins A and B that demonstrate anti-diabetes, anti-inflammatory, and anti-oxidative stress [13,14,15,16], it has been revealed that interruptins A and B effectively promoted glucose consumption to hepatocytes and adipocytes via modulation of the PPAR pathway combined with upregulation of the Slc2a1/Glut1 gene and GLUT2 and GLUT4 proteins [15,16]. The gene discussed is SLC2A1; the disease is diabetes mellitus.