Our findings revealed that the expression of bax (a pro-apoptotic factor), cleaved caspase 3 and c-PARP proteins were substantially increased, while the expression of bcl2 (an anti-apoptotic factor) was decreased in both MCF-7 and MDA-MB-231 breast cancer cell lines by azilsartan leading to the induction of apoptosis, as summarized in Figure 11, which was also confirmed by the flow cytometric analysis. The gene discussed is CASP3; the disease is breast carcinoma.