The accumulation of β-catenin in the cell nucleus triggers mutations, driving the transcription of oncogenes, such as Jun, Axin-2, c-Myc and Cyclin-D1, resulting in the initiation and progression of various types of carcinoma, including colon cancer, hepatocellular carcinoma, pancreatic cancer, lung cancer and ovarian cancer [14,15]. This evidence concerns the gene MYC and colonic neoplasm.