When looking at ILC populations in the peritoneal cavity after i.p. administration of T. gondii, interestingly, Snyder et al. found that ILC1s showed a noticeable increase in the proliferation marker Ki67 after infection, which indicates that ILC1s rather proliferate in situ, than are recruited from other parts at mucosal tissues of the body [105]. Here, CCL27 is linked to infection.