Klose et al. demonstrated that mice deficient for T-bet secrete less IFNγ due to an ILC1 deficiency, whereas NK cell numbers remained the same in the small intestine during T. gondii infection, suggesting that ILC1 are a main source of IFNγ in the small intestine during infection, apart from NK cells or NKp46+ NK1.1+ ILC3s [99,100]. Here, NCR1 is linked to infection.