During atherosclerosis, in response to the signals, such as monocyte chemoattractant protein-1 (MCP-1) chemokine (C-C motif) ligands 3, 4, and 5 (CCL3, CCL4, CCL5) released from the endothelial cells damaged by the accumulating lipoproteins, monocytes migrate to the damaged subendothelial sites in the attempt to remove oxLDL. Here, CCL2 is linked to atherosclerosis.