However, these findings were obtained in healthy cells including fibroblasts, bone marrow, and kidney cells; while data in cancer cells showed that overexpression of GSK-3β activated mTOR complex-1 (mTORC1) leading to activation of the mTOR pathway, indicating that the GSK-3β/mTOR interaction in cancer cells is different from normal cells [33]. The gene discussed is MTOR; the disease is cancer.