However, tetra peptide N-acetyl-seryl-aspartyl-lysyl-proline (Ac-SDKP) attenuated silicosis-induced rat lung fibrosis and blocked AngII-induced myofibroblast differentiation by restoring acetylated α-tubulin, suggesting a positive role of acetylated α-tubulin in pulmonary fibrosis treatment [28]. The gene discussed is AGT; the disease is silicosis.