CXCR3 and synovial sarcoma: In addition to the pro-inflammatory cytokines produced by effector T cells, T cell trafficking into the disease-target salivary glands is also a vital pathogenic event of SS, which requires the interaction of T cell-expressed chemokine receptors, such as CXCR3 and their specific ligands CXCL9, CXCL10 and CXCL11 [15].