Interestingly, the FSHB gene polymorphisms can be involved in the biology of endometriosis not only due to the above-mentioned FSH pathways, but also due to the effects of ARL14EP, the expression of which they control (according to our in silico models, endometriosis high risk alleles T of rs11031002 and rs11031005 are linked with low expression of ARL14EP). This evidence concerns the gene BRD2 and endometriosis.