At the same time, significant phenotypic effects of the interaction of these polymorphisms with respect to endometriosis can manifest themselves in the interaction of sex hormones whose levels they determine, such as LH [16], serum levels of protein CGA, FSHB [19], (rs11031002 FSHB), FSH [16], total and bioavailable testosterone levels [17], (rs11031005 FSHB), and oestradiol (rs117585797 ANO2 [16]). The gene discussed is ANO2; the disease is endometriosis.