Expanding on the potential molecular mechanisms of isothiocyanates from the extracts (AITC and BITC) linked to colorectal cancer, the in silico analysis agreed with the reported effects of BITC on the induction of IκB kinase phosphorylation, IκB-a, and p65, allowing NF-kB translocation into the nucleus [67]. This evidence concerns the gene NFKB1 and colorectal cancer.