Yang et al. [94] showed that lncRNA H19 expression was significantly increased in both LPS- and lipoteichoic acid (LTA)-induced inflammatory MAC-T cells, and the overexpression of lncRNA H19 promoted TGF-β1-induced EMT and the overaccumulation of extracellular matrix (ECM) proteins, which led to the formation of breast fibrosis (Figure 8). Here, H19 is linked to breast fibrosis.