In addition, in a sepsis-induced lung injury model, Qiu et al. [86] detected a low expression of lncRNA TUG1, and overexpression of lncRNA TUG1 ameliorated sepsis-induced lung injury, secretion of proinflammatory cytokines, and apoptosis by suppressing miR-34b-5p and promoting GRB2-associated binding protein 1 (GAB1). The gene discussed is GAB1; the disease is Sepsis.