“Cytokine storm”, mostly detected in the peripheral blood of COVID-19 patients [3], runs with high levels of components such as interferons (IFNs), chemokines, interleukin (IL)-6, IL-8, IL-1β, and chemokines, along with reactive oxygen species, which are harmful by inducing cellular necrosis, diffusing alveolar lesion, pulmonary fibrosis, and fibrin deposition [4,5]; thus, alleviating hyperinflammation is crucial to improving prognosis. This evidence concerns the gene CXCL8 and COVID-19.