Therefore, our result, conforming with previous reports, indicates that simultaneous upregulation of CD40, Flt3L, IL-6, IL-8, and LIF (Figure 3), in association with CAPG and EDIL3 (Figure 3), plays a pivotal role in inflammation and recruitment of immune cells in lung tissues of COVID-19 patients. The gene discussed is FLT3LG; the disease is COVID-19.