ING5 inhibits proliferation, glucose catabolism, anti-apoptosis, migration, invasion, epithelial- mesenchymal transition (EMT) or lung metastasis, and induces cell arrest, senescence, autophagy, fat accumulation, and chemoresistance in gastric, colorectal, breast, lung, ovarian cancer cells, neuroblastoma and glioma cells (Gou et al., 2015; Zhao et al., 2016; Ding et al., 2017; Zhao et al., 2017; Zheng et al., 2017; Wu et al., 2018; Yu et al., 2022; Zheng et al., 2022). This evidence concerns the gene ING5 and ovarian carcinoma.