SOAT1 and inborn error of immunity: Cytokine–cytokine receptor interaction, chemokine signaling pathway, NK cell-mediated cytotoxicity, oxidative phosphorylation, systemic lupus erythematosus, JAK–STAT signaling pathway, cell adhesion molecule cams, T cell receptor signaling pathway, Toll-like receptor (TLR) signaling pathway, proteasome, and primary immunodeficiency were enriched in the low-risk subgroups.