Chemokines such as CXCL8/interleukin-8 (IL-8), CXCL10/interferon gamma-induced protein 10 (IP-10), CCL2/monocyte chemoattractant protein 1 (MCP-1), CXCL9/interferon gamma-induced monokine (MIG), and CCL5/RANTES lead to the activation and recruitment of leukocytes to the site of infection, resulting in the potentiation of the inflammatory response and its termination [23,25,26,27,28]. The gene discussed is CCL2; the disease is infection.