They were mainly enriched in pancreatic cancer; Kaposi sarcoma-associated herpesvirus infection; hepatitis B; autophagy—animal; pathways in cancer; human cytomegalovirus infection; hepatitis C; lipid and atherosclerosis; Human T-cell leukemia virus 1 infection; FoxO signaling pathway; MAPK signaling pathway; Shigellosis; Epstein-Barr virus infection; proteoglycans in cancer; chemical carcinogenesis—reactive oxygen species; microRNAs in cancer; PI3K-Akt signaling pathway. This evidence concerns the gene AKT1 and hepatitis B virus infection.