MET functions upstream of numerous signaling pathways promoting tumorigenesis including PI3K/AKT/mTOR, STAT, MEK/ERK, GRB2, GAB1 and RAC1 thus promoting cell survival, proliferation, angiogenesis and mobility and being similar to the tumorigenesis found in certain forms of hepatocellular carcinoma [84]. Here, MET is linked to hepatocellular carcinoma.