As expected, the UVC/PHA-triggered oxidative stress bursts may have affected oxidative-stress-associated responses, as shown by the upregulation of γH2AX- and 8-OHdG-type DNA damage and caspase 3/7-indicated apoptosis in oral cancer cells compared to the separate treatments (Figure 8, Figure 9 and Figure 4), respectively. The gene discussed is CASP3; the disease is lip and oral cavity carcinoma.