KLF6 and ischemic stroke: The lncRNA Hox transcript antisense intergenic RNA (HOTAIR) can competitively bind miRNA-148a-3p through a competing endogenous RNA mechanism to upregulate Kruppel-like factor 6 (KLF6) expression, inhibit the signal transducer and activator of transcription 3 pathway, promote cell apoptosis and the inflammatory response, and aggravate nerve injury after ischemic stroke [123].