In other research, it was shown that the cross of HSA-LR with the mouse model with multisystemic overexpression of MBNL1 or overexpression restricted just to the skeletal muscles (MBNL1-OE) showed the rescue of DM-like defects, including the decrease of percentage of fibers with central nuclei reflecting myopathy changes. This evidence concerns the gene MBNL1 and myopathy.