XCR1 and neoplasm: viSNE analysis revealed that NDV-killed tumor cells preferentially induced activation of Lin-I-Ad+CD11c+ conventional DCs (cDCs), as indicated by upregulation of MHC II, co-stimulatory molecules (CD86, CD40) and PDL1 (Fig. 2a), with similar effects in both cross-presenting XCR1+ cDC1 and CD11b+ cDC2 subsets (Supplementary Fig. 2a).