C5‐deficient, but not C6‐deficient, mice were protected, and, in a related system where antibody to GBM nephritis was induced in FH knockout mice, mice had longer and persistent glomerular infiltration with neutrophils compared to control animals, and inhibition of C5 by a monoclonal antibody provided anti‐inflammatory protection.71 The gene discussed is C5; the disease is glioblastoma.