ABCA1 and atherosclerosis: In the present study, we provide data demonstrating that miR-199a-5p and ABCA1 are inversely regulated during hypoxia conditions, and that miR-199a-5p represents a novel posttranscriptional regulator of ABCA1 by in primary mouse macrophages and cholesterol-laden macrophages which pinpoint a possible role in atherosclerosis.