The various hepatic manifestations are hepatomegaly; steatosis; elevated liver enzymes alanine aminotransferase (ALT), aspartate aminotransferase (AST), and gamma-glutamyl transferase (GGT); hyperlipidaemia; hypertriglyceridemia; and liver failure. Further, it is noticed that hypoperfusion, hypoxia, sepsis, hypermetabolic states, and vasopressor therapy involved in this syndrome also cause impairment of liver function and exaggerate hyperlipidaemia [26,41]. The gene discussed is GPT; the disease is Hepatic failure.