In particular, some miRNAs were characterized for their inhibitory effect on fibrosis: miR-15 was described to suppress inflammation and fibrosis of MCs through VEGF inhibition [26]; miR-30a negatively regulated TGF-β1-induced EMT and peritoneal fibrosis by targeting Snai1 [27]; miR-9-5p suppressed pro-fibrogenic transformation of fibroblasts and MCs from PD patients [28]; miR-302c modulated peritoneal dialysis-associated fibrosis by targeting CTGF [29]; miR-29b inhibited peritoneal fibrosis in a mouse model of PD [30]. This evidence concerns the gene TGFB1 and kidney failure.