evaluated the relationship between core clock genes and hepatitis C virus (HCV)‐related viral infection in two in vitro models, the human hepatoma Huh‐7 and the non‐human primate OR6 cell lines and found that PER2 overexpression decreased HCV RNA replication, suggesting that PER2 can act as an inhibiting factor of viral replication.94 This evidence concerns the gene CLOCK and viral infectious disease.