In the present study, genotyping the APOBEC3A/B deletion polymorphism in 1470 endometrial cancer (EC) patients, we also found the genotype distribution to be in Hardy–Weinberg (H–W) equilibrium (p = 0.78) with 1264 (85.9%) homozygous for the insertion allele, 199 (13.5%) heterozygous, and seven (0.48%) homozygous for the deletion allele (Table 1), resulting in a MAF of 0.072. The gene discussed is APOBEC3A; the disease is endometrial cancer.