SNCA and Parkinson disease: We thus wondered whether CoCl2 could promote beneficial effects in C. elegans strains expressing human aggregation-prone proteins, poly-Q40 and α-synuclein (under muscle-specific promote, unc-54), and widely exploited as model systems to respectively study Huntington’s and Parkinson’s diseases (Alexander et al., 2014; Caldwell et al., 2020; Liang et al., 2020).