For instance, the mutated KCNQ2 and KCNQ3 genes, which encode Kv7.2 and Kv7.3, respectively, enable benign familial neonatal convulsions (Maljevic and Lerche, 2014), and genetic suppression of small-conductance Ca2+-activated K+ channels (SK) in deep cerebellar neurons can result in ataxia (Lam et al., 2013). Here, KCNQ2 is linked to cerebellar ataxia.