The identification of eNAMPT, as a target and biomarker in sarcoidosis is consistent with the top significant dysregulated pathways identified comprised of cytokine-cytokine receptor interaction, MAPK signaling, IL-17, Jak-STAT signaling, chemokine signaling, NF-kappa B, and TNF signaling pathways (Figure 5). This evidence concerns the gene IL17A and sarcoidosis.