Specifically, it has been demonstrated that factors derived from tumors induce skeletal muscle atrophy by activating NF-kB and proteolysis of specific muscle mediated by ubiquitin proteasome pathway (UPS) in studies with mouse Lewis lung carcinoma (LLC) and C26-colon carcinoma (C26) (18). Here, NFKB1 is linked to Carcinoma, Lewis Lung.