KHSRP and neoplasm: Ebv-circLMP2A interacted with KHSRP to increase the KHSRP-mediated degradation of VHL mRNA, resulting in an accumulation of HIF1 under hypoxia, which was crucial in controlling tumor angiogenesis in EBVaGC and might be a good therapeutic target for EBVaGC (Du et al., 2022).