A subset of COVID-19-treated CHD patients had myocardial injury, as evidenced by changes in the electrocardiogram and increased levels of troponin, creatine kinase (CK), creatine kinase-MB (CK-MB), and the N-terminal fragment of brain natriuretic peptide (BNPP) (NT-Pro-BNP) in about 8-12% of patients (6–8). Here, NPPB is linked to coronary artery disorder.