DRAK1 inhibits the growth and metastasis of advanced cervical cancer cells through two pathways: interfering with the homo-oligomerization of tumor necrosis factor (TNF) receptor-associated factor 6 (TRAF6) and specifically reducing the stability of TRAF6 protein through an autophagy-mediated degradation pathway (Park et al., 2020). Here, TRAF6 is linked to cervical carcinoma.