To further investigate the mechanism of BLWE promoting neurogenesis in the treatment of AD, we analyzed the expression of proteins such as t-GSK-3β, p-Tyr216-GSK-3β, t-β-catenin, p-β-catenin, and CylinD1, which were related to the Wnt/β-catenin signaling pathway (Figure 7A,B). The gene discussed is GSK3B; the disease is Alzheimer disease.