As a key factor in lipid metabolism, CPT1A is engaged in the management of lipid metabolism through FAO in colorectal cancer [25], breast cancer [26], nasopharyngeal carcinoma [27], etc. Recently, emerging evidence has shown CPT1A is downregulated in ccRCC, concomitant with suppressed FAO; at the same time, overexpression of CPT1A reduces lipid accumulation in ccRCC, thus suppressing cell proliferation [28, 29]. Here, CPT1A is linked to nasopharyngeal carcinoma.