CCL28 was a favorable prognostic factor for luminal-like cases and unfavorable for triple-negative breast cancer [36], inhibited oral squamous cell carcinoma by CCL28/RARβ [37], promoted angiogenesis in lung adenocarcinoma by CCL28/CCR3 signaling pathway [38], and even was induced to enhance the recruitment of Treg under hypoxia conditions [39]. This evidence concerns the gene CCR3 and triple-negative breast carcinoma.