IUGR leads to a reduced density of glutamic acid decarboxylase 67 (GAD67)‐, calbindin‐, and calretinin (CR)‐immunoreactive (IR) neurons within the primary somatosensory cortex, but an increased density of GAD67‐ and CR‐IR neurons in the prefrontal cortex of adult rats (Delcour et al., 2012). The gene discussed is GAD1; the disease is fetal growth restriction.