ITGAM and viral myocarditis: To summarize, our findings suggest that (I) cardiac involvement in COVID-19 does not present as conventional viral myocarditis defined by mononuclear infiltrates and myocyte damage, but rather as a perivascular infiltration by a special subpopulation of CD11b + /TIE2 + macrophages contributing to cardiac neoangiogenesis and remodeling of the fibrovascular interface.