For example, IFN-stimulated genes and pathways were identified in the comparison of our allergic asthma versus controls, as well as in the obesity-associated low type-2 asthma cohort; however, in contrast to obesity-associated asthma, we did not identify gap junction pathways or its related genes [72]. This evidence concerns the gene IFNA1 and obesity due to melanocortin 4 receptor deficiency.