Differential protein expression patterns were analyzed by bioinformatics and are displayed in this report according to association with the (i) dystrophin-glycoprotein complex, (ii) the established marker signature of dystrophinopathy, (iii) excitation-contraction coupling, (iv) the annexin family, (v) the collagen family, and (vi) the extracellular matrix. This evidence concerns the gene DMD and neuromuscular disease caused by qualitative or quantitative defects of dystrophin.