After comparing the concentrations of ICAM-1, VCAM-1, P-selectin and fractalkine in convalescents to those in healthy controls, the authors concluded that SARS-CoV-2 infection in the past does not impose an increased risk of cardiovascular events, not only for patients with a mild form of infection, but also for those who were severely ill [41]. This evidence concerns the gene ICAM1 and infection.