Wei et al. reported that Ac-SDKP was downregulated in a CCl4-treated liver fibrosis rat model, and exogenous Ac-SDKP caused the decreased expressions of Wilms’ tumor 1-associated protein (WTAP) and N6-methyladenosine (m6A), which suppressed the Hh signaling, alleviated the HSCs apoptosis and fibrosis [50]. This evidence concerns the gene WTAP and Hepatic fibrosis.