TNNI3 and cerebrovascular disorder: This might be explained by the fact that the traditional myocardial injury markers, CK-MB and cTnI, were passively and non-specifically released into extracellular spaces in response to cardiac ischemic injuries and other factors such as global asphyxiation, hyperthermia, cerebrovascular disease, carbon monoxide intoxication, electrocution, fatal methamphetamine abuse, and psychotropic drug intoxication [41,42,43].