Mechanistically, these antitumor effects of SST and CORT were associated with the alteration in the levels of critical genes and oncogenic signaling pathways that have been reported to be frequently associated with the functional and cellular control of the SST system in multiple ERCs (e.g., proliferation, migration, and PCa-aggressiveness features) [14,15,18,20,28,31,32]. Here, SST is linked to posterior cortical atrophy.