TGM 2 was previously mentioned regarding the deep association causing an allergic status, including IL-33 expression [8], phospholipase A2 (PLA2) expression in asthma [9], and allergic airway inflammation driven by a steroid-resistant cascade of Wnt5a, TGM 2, and leukotrienes [10], but our results revealed that TGM 2 playing a critical role of both inducer of allergic condition and wound healing was not deeply associated with NP development and/or maintenance in terms of the main protein catalyzation factor. Here, WNT5A is linked to asthma.